David Karp, M.d., Ph.d.
Assistant Professor
immunology
UT Southwestern Medical Center
Bangladesh
Biography
Dr. Karp was raised near Cleveland, OH. He obtained his bachelor’s degrees from the Massachusetts Institute of Technology and his medical and doctoral degrees from Washington University in St. Louis. He also completed his residency and clinical fellowship training at Barnes Jewish Hospital in St. Louis before joining the NIH as a research fellow. He has been on the faculty of UT Southwestern since 1991. For the past 25 years, Dr. Karp has studied the human immune system in health and disease. This has involved research on the genes, proteins, and cells that control immunity to harmful pathogens, as well as lead to disease states like systemic lupus erythematosus and systemic sclerosis. His most recent studies have focused on the concept of pre-clinical immunity – the transition from people from a healthy state to having an activated immune system without symptoms to having genuine symptoms of autoimmune disease. Understanding this transition may lead to better diagnostic tools and the ability to treat autoimmune diseases at an
Research Interest
Dr. Karp was raised near Cleveland, OH. He obtained his bachelor’s degrees from the Massachusetts Institute of Technology and his medical and doctoral degrees from Washington University in St. Louis. He also completed his residency and clinical fellowship training at Barnes Jewish Hospital in St. Louis before joining the NIH as a research fellow. He has been on the faculty of UT Southwestern since 1991. For the past 25 years, Dr. Karp has studied the human immune system in health and disease. This has involved research on the genes, proteins, and cells that control immunity to harmful pathogens, as well as lead to disease states like systemic lupus erythematosus and systemic sclerosis. His most recent studies have focused on the concept of pre-clinical immunity – the transition from people from a healthy state to having an activated immune system without symptoms to having genuine symptoms of autoimmune disease. Understanding this transition may lead to better diagnostic tools and the ability to treat autoimmune diseases at an
