Jennifer corcoran
Assistant Professor
Department of Microbiology and Immunology
Dalhousie University
Canada
Biography
Dr. JENNIFER CORCORAN is affiliated to Department of Microbiology and Immunology, Dalhousie University. Dr. JENNIFER CORCORAN is currently providing services as Assistant Professor. Dr. JENNIFER CORCORAN has authored I145and co-authored multiple peer-reviewed scientific papers and presented works at many national and International conferences. Dr. JENNIFER CORCORAN contributions have acclaimed recognition from honourable subject experts around the world. Dr. JENNIFER CORCORAN is actively associated with different societies and academies. Dr. JENNIFER CORCORAN academic career is decorated with several reputed awards and funding. Dr. JENNIFER CORCORAN research interests include Ten to fifteen percent of all human cancers are caused by viruses; yet the precise mechanisms of viral oncogenesis remain poorly understood. We study how the virus, Kaposi’s sarcoma-associated herpesvirus (KSHV) uses specific virally-encoded oncoproteins to deregulate normal endothelial cells and cause the cancer, Kaposi’s Sarcoma (KS)..
Research Interest
Ten to fifteen percent of all human cancers are caused by viruses; yet the precise mechanisms of viral oncogenesis remain poorly understood. We study how the virus, Kaposi’s sarcoma-associated herpesvirus (KSHV) uses specific virally-encoded oncoproteins to deregulate normal endothelial cells and cause the cancer, Kaposi’s Sarcoma (KS).
Publications
-
Corcoran, J. A., H. A. Saffran, B. A. Duguay and J. R. Smiley. 2009. Herpes simplex virus UL12.5 targets mitochondria through a mitochondrial localization sequence proximal to the N-terminus. J. Virol. 83:2601-10.
-
Corcoran, J.A., D. Khaperskyy, B. P. Johnston, C.A. King, D.P. Cyr, A.V. Olsthoorn, and C. McCormick. 2012. Kaposi’s sarcoma-associated herpesvirus G-protein coupled receptor prevents AU-rich element-mediated decay. J. Virol. 86(16): 8859-8871.
-
Corcoran, J.A., B. P. Johnston, and C. McCormick. 2014. Viral activation of MK2-hsp27-p115RhoGEF-RhoA signaling axis causes cytoskeletal rearrangements, p-body disruption and ARE-mRNA stabilization. PLOS Pathogens
