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Diana Anderson

Professor
School of Chemistry and Biosciences
Ahmedabad University, Ahmedabad
India

Biography

Professor, Established Chair and Research Co-ordinator in the Department of Biomedical Sciences, University of Bradford, Bradford, UK, 2000 – to date Head of Educational Services, TNO BIBRA International, 1999 - 2000 Senior Associate, Co-ordinator of External Affairs, BIBRA International, 1992 - 1999 Assistant Director and Co-ordinator of Group Forum/External Affairs, BIBRA International 1987 - 1992 Research Manager for Genetic and Reproductive Toxicology, BIBRA, 1987-2000 Head of Genetic and Reproductive Toxicology, BIBRA, 1981 - 1987 Head of Mutagenesis Studies at Central Toxicology Laboratory, ICI Ltd, Alderley Park, Cheshire, UK 1974 - 1981 Research worker - Paterson Laboratories, Christie Hospital, University of Manchester, UK 1970 - 1974 Lecturer in Biology, Didsbury Training College, Manchester, UK 1969 - 1970 Research Assistant, Department of Cancer Studies, Leeds University, UK 1968 - 1969 Tutor in School of Biological Sciences, Sydney University, New South Wales, Australia, 1965 - 1968 Lecturer, Llandaff Technical College, Cardiff, UK, 1963 - 1964.

Research Interest

Cancer, oxygen radicals, human biomonitoring, unbalanced precursor pools, dietary imbalance, male-mediated developmental defects, nanotoxicology, general toxicology

Publications

  • Ruf A A, Cuthbert A C, Foroutan B and Anderson D (2006). p53 Expression in haematological malignancies: Response to alkylating agent in the Comet Assay Journal of Preventive Medicine. 14 (1-2) 32-45

  • Schallreuter K U, G.Chuichiarelli, E Cemeli, SM Elwary, JM Gillbro, J D Spencer, H Rokos, A Panske, B Chawan, JM wood and D Anderson (2006). Oestrogens can contribute to hydrogen peroxide generation and quinine–mediatedDNA damage in peripheral blood lymphocytes from patients with vitiligo. Journal of Investigative Dermatology 126 1036-1042

  • Cemeli E, Wagner ED, Anderson D, Richardson SD and Plewa MJ (2006). Modulation of the cytotoxicity and genotoxicity of the drinking water disinfection by-products iodoacetic acid by suppression of oxidative stress. Environmental Science and Technology 40 1878-1883

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