Masaki Imanishi
Assistant Professor
Faculty of Medicine
University of Tokushima
Japan
Biography
Masaki Imanishi has done his Doctor of Medicine from Tokushima University in 2014. Currently he is working as Assistant Professor in Faculty of Medicine in Tokushima University. His research interests are Pharmacology, Cardiovascular Pharmacology, Clinical Pharmacology, Molecular Pharmacology. Several papers of his work were published in reputed international journals. He is the member of The Japanese Pharmacological Society, The Pharmaceutical Society of Japan, The Japanese Society of Clinical Pharmacology and Therapeutics, American Heart Association, Japan Society of Hypernatremia, Japan Society of Rehabilitation Society, Japan Society of Cardiovascular Activity Materials, Japan Medical and Pharmaceutical Society, Japan Hospital Pharmacists Association, Tokushima Prefectural Hospital Pharmacists Association, Japan Society for the Promotion of Science.
Research Interest
Pharmacology, Cardiovascular Pharmacology, Clinical Pharmacology, Molecular Pharmacology
Publications
-
Koyama Satoshi, Matsunaga Shinji, Masaki Imanishi, Yoichi Maekawa, Kitano Hiroya, Takeuchi Hiromi and Shuhei Tomita. Tumour blood vessel normalisation by prolyl hydroxylase inhibitor repaired sensitivity to chemotherapy in a tumour mouse model. Scientific Reports. 2017, 7: 45621.
-
Hirofumi Hamano, Yasumasa Ikeda, Hiroaki Watanabe, Yuya Horinouchi, Yuki Izawa-Ishizawa, Masaki Imanishi, Yoshito Zamami, Kenshi Takechi, Licht Miyamoto, Keisuke Ishizawa, Koichiro Tsuchiya and Toshiaki Tamaki. The uremic toxin indoxyl sulfate interferes with iron metabolism by regulating hepcidin in chronic kidney disease. Nephrology, Dialysis, Transplantation. 2017.
-
Yasumasa Ikeda, Yuya Horinouchi, Hamano Hirofumi, Hirayama Tasuku, Seiji Kishi, Yuki Izawa-Ishizawa, Masaki Imanishi, Yoshito Zamami, Kenshi Takechi, Licht Miyamoto, Keisuke Ishizawa, Ken-ichi Aihara, Hideko Nagasawa, Koichiro Tsuchiya and Toshiaki Tamaki. Dietary iron restriction alleviates renal tubulointerstitial injury induced by protein overload in mice. Scientific Reports. 2017, 7(1): 10621.
