Laundry González, Sergio

Biography

Sergio Lavandero González was trained at the National Institute and the University of Chile. He has a track record of more than 30 years in biomedical research. He graduated as a Pharmaceutical Chemist from the University of Chile in 1983. He later obtained a PhD in Biochemistry from the University of Chile in 1993, with a PhD grant from the Andes Foundation. His knowledge in the area of ​​signal transduction and cardiovascular system achieved through his postdoctoral stays in Imperial College of Science, Technology & Medicine (England); Erasmus Universitat (The Netherlands); National Institutes of Health (USA) and St. Vincent's Hospital, Research Cardiovascular Center (Australia). In 1985 he entered as an instructor to the University of Chile, reaching the rank of Full Professor after 15 years. He is currently Professor of the Department of Biochemistry and Molecular Biology of the Faculty of Chemical and Pharmaceutical Sciences and the Program of Cellular and Molecular Biology of the Institute of Biomedical Sciences of the Faculty of Medicine. Dr. Lavandero is Adjunct Professor of the University of Texas Southwestern Medical Center (Dallas, USA) between 2014-2017. He has taught undergraduate in the Faculties of Chemical and Pharmaceutical Sciences, Medicine and Sciences) for the careers of Biochemistry, Chemistry-Pharmacy, Medicine and Engineering in Biotechnology. He has directed 66 undergraduate papers. It participates actively in 7 Doctorate Programs of the University of Chile (Biochemistry, Pharmacology, Cellular and Molecular Biology, Neurosciences, Pharmaceutical Sciences, Biotechnology and Biomedical Sciences) forming part of its cloisters. He has directed 14 postdocs, 69 doctoral theses and 8 master's theses.

Research Interest

Transduction of signals in the cardiovascular system, specifically the molecular mechanisms that regulate death, survival and metabolism of the heart, key processes in the development of pathologies such as myocardial infarction, heart failure and arterial hypertension and The molecular basis of damage mechanisms of type 2 diabetes mellitus in cardiac and skeletal muscle.