Dr Steve Jacques
Senior Lecturer
Department of Medical Education
University of Leicester
United Kingdom
Biography
Steve originally trained in clinical medicine at the University of Birmingham, and worked for two years at the Queen Elizabeth Hospital in a variety of medical specialties. Following foundation training he decided to continue the research that he had pursued during an intercalated year and undertook a PhD in Ann Logan and Martin Berry's lab, where he developed novel gene therapeutic approaches for spinal cord injury. After his PhD, Steve became an anatomy demonstrator in Birmingham, which he did for a further three years. He then became Lecturer in Human Biology, Biochemistry and Disease at the School of Biosciences in Birmingham before joining the University of Leicester as Senior Lecturer in 2016. In 2015, Steve won an Outstanding Teaching Award at Birmingham for his contributions to the Human Biology programme. Steve originally trained in clinical medicine at the University of Birmingham, and worked for two years at the Queen Elizabeth Hospital in a variety of medical specialties. Following foundation training he decided to continue the research that he had pursued during an intercalated year and undertook a PhD in Ann Logan and Martin Berry's lab, where he developed novel gene therapeutic approaches for spinal cord injury. After his PhD, Steve became an anatomy demonstrator in Birmingham, which he did for a further three years. He then became Lecturer in Human Biology, Biochemistry and Disease at the School of Biosciences in Birmingham before joining the University of Leicester as Senior Lecturer in 2016. In 2015, Steve won an Outstanding Teaching Award at Birmingham for his contributions to the Human Biology programme.
Research Interest
Although Steve does not have an active research group, he is always happy to contribute to research activities in the fields of anatomy (particularly neuroanatomy), neural regeneration and education.
Publications
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Ahmed Z, Jacques SJ, Berry M, Logan A (2009) Epidermal growth factor receptor inhibitors promote CNS axon growth through off-target effects on glia. 36:142-150.