Venugopal Venna
Assistant Professor
Department of Neurology
The University of Texas Health Science Center
United States of America
Biography
Dr. Venna obtained his Bachelor’s Degree in Pharmacy from Gulbarga University, India. He received his PhD in 2009 from the University of Lille-2, France. Much of his doctoral work focused on behavior and the response to pharmacological agents for the treatment of depression. After completing his Ph.D., Dr. Venna joined Dr. McCullough’s Cerebrovascular Research Group to obtain his post-doctoral training in ischemic stroke research. He was subsequently appointed as faculty at the University of Connecticut Health Center. In 2015, Dr. Venna joined the Department of Neurology at UTHealth in Houston, TX. Because of his long-standing interest in basic and translational stroke research, Dr. Venna and his research team have established translationally relevant stroke models in rodents. These models mimic the human disease and allow for the investigation and elucidation of the underlying mechanisms that determine acute and long-term outcomes after stroke. Dr. Venna has published his research findings in several prestigious journals, including Stroke, Neuroscience, Translational Psychiatry, Psychoneuroendocrinology, Acta Neuropathologica and the European Journal of Neuroscience. He received a number of awards and honors, including the American Heart Association Young Investigator Travel Award, Founders Affiliate Grants from AHA and the French Pharmacological Society Fellowship Award. His research is currently supported by Scientist Development Grant from the American Heart Association.
Research Interest
Stroke, Traumatic Brain Injury (TBI), Depression, Acute and Chronic Stress
Publications
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Du E, McAllister P, Venna VR, Xiao L. Clinically Relevant Concentrations of Ketamine Inhibit Osteoclast Formation In Vitro in Mouse Bone Marrow Cultures. J Cell Biochem. 2017 Apr;118(4):914-923.
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Adnan S, Nelson JW, Ajami NJ, Venna VR, Petrosino JF, Bryan RM Jr, Durgan DJ. Alterations in the gut microbiota can elicit hypertension in rats. Physiol Genomics. 2017 Feb 1;49(2):96-104.
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Verma R, Cronin CG, Hudobenko J, Venna VR, McCullough LD, Liang BT. Deletion of the P2X4 receptor is neuroprotective acutely, but induces a depressive phenotype during recovery from ischemic stroke. Brain Behav Immun. 2017 Jul 24. pii: S0889-1591(17)30384-7.
