Reproductive Medicine
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Natalia Gokina

Associate Professor
Department of Obstetrics, Gynecology & Reproductive Sciences
University of Vermont
United States of America

Biography

Prof/Dr Natalia Gokina has received his PhD Ukranian Academy of Sciences, Kiev, Ukraine, Physiology. Currently, she is working as Professor in university of vermont. She has successfully completed her Administrative responsibilities as Professor. Her research has includedcardiovascular physiology, specifically, control of vascular smooth muscle and endothelial cell calcium and membrane potential during pregnancy, and in the signal transduction mechanisms underlying arterial reactivity. Current research in Dr. Gokina’s lab is focused on understanding cellular and molecular mechanisms of uteroplacental vascular dysfunction in diabetic pregnancy.

Research Interest

Dr. Gokina’s primary research interest is in cardiovascular physiology, specifically, control of vascular smooth muscle and endothelial cell calcium and membrane potential during pregnancy, and in the signal transduction mechanisms underlying arterial reactivity. Current research in Dr. Gokina’s lab is focused on understanding cellular and molecular mechanisms of uteroplacental vascular dysfunction in diabetic pregnancy.

Publications

  • Gokina NI, Knot HJ, Nelson MT, Osol G. Increased Ca 2+ sensitivity as a key mechanism of PKC-induced constriction in pressurized cerebral arteries. American Journal of Physiology-Heart and Circulatory Physiology. 1999 Sep 1;277(3):H1178-88.

  • Gokina NI, Park KM, McElroy-Yaggy K, Osol G. Effects of Rho kinase inhibition on cerebral artery myogenic tone and reactivity. Journal of Applied Physiology. 2005 May 1;98(5):1940-8.

  • Osol G, Brekke JF, McElroy-Yaggy K, Gokina NI. Myogenic tone, reactivity, and forced dilatation: a three-phase model of in vitro arterial myogenic behavior. American Journal of Physiology-Heart and Circulatory Physiology. 2002 Dec 1;283(6):H2260-7.

  • CIPOLLA MJ, GOKINA NI, OSOL G. Pressure-induced actin polymerization in vascular smooth muscle as a mechanism underlying myogenic behavior. The FASEB Journal. 2002 Jan 1;16(1):72-6.

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